منابع مشابه
Cordotomy for Thalamic Pain
Mr. E., a paper-hanger, married and father of one child, suffered a gonorrheal infection in 1919. In 1929, when he was 46 years of age, he was troubled one evening with a pain of moderate severity in his right hand and forearm. The following morning there was a continuous dull ache in this hand and forearm and at least every half hour a spasm of severe pain which would last for several minutes....
متن کاملSymbolic Aspects of Thalamic Pain
Pain associated with hemiplegia has been considered mainly in the context of the thalamic syndrome. Since the original observations by Dejerine' and Roussy,' many cases have been reported and all textbooks of neurology carry a description of it. The cardinal points have been the presence, on the side opposite the lesion, of spontaneous pain and an over-reaction to sensory stimuli. Most of the c...
متن کاملThalamic mechanism of pain: shell theory of thalamic nociception.
In both cats and Japanese macaques, there are nociceptive specific (NS) and wide dynamic range (WDR) neurons in the shell region of the caudal ventrobasal (VB) complex of the thalamus. This comprises both the nucleus ventralis posteromedialis proper (VPM proper), and the nucleus ventralis posterolateralis (VPL). These two classes of nociceptive neurons are spatially segregated, WDR neurons bein...
متن کاملThalamic Pain Misdiagnosed as Cervical Disc Herniation.
Thalamic pain is a primary cause of central post-stroke pain (CPSP). Clinical symptoms vary depending on the location of the infarction and frequently accompany several pain symptoms. Therefore, correct diagnosis and proper examination are not easy. We report a case of CPSP due to a left acute thalamic infarction with central disc protrusion at C5-6. A 45-year-old-male patient experiencing a ti...
متن کاملPain without Nociceptors? Nav1.7-Independent Pain Mechanisms
Nav1.7, a peripheral neuron voltage-gated sodium channel, is essential for pain and olfaction in mice and humans. We examined the role of Nav1.7 as well as Nav1.3, Nav1.8, and Nav1.9 in different mouse models of chronic pain. Constriction-injury-dependent neuropathic pain is abolished when Nav1.7 is deleted in sensory neurons, unlike nerve-transection-related pain, which requires the deletion o...
متن کاملذخیره در منابع من
با ذخیره ی این منبع در منابع من، دسترسی به آن را برای استفاده های بعدی آسان تر کنید
ژورنال
عنوان ژورنال: PAIN RESEARCH
سال: 1992
ISSN: 0915-8588,2187-4697
DOI: 10.11154/pain.7.1